ΔNp63 in squamous cell carcinoma: defining the oncogenic routes affecting epigenetic landscape and tumour microenvironment

Mol Oncol. 2019 May;13(5):981-1001. doi: 10.1002/1878-0261.12473. Epub 2019 Mar 22.

Abstract

Squamous cell carcinoma (SCC) is a treatment-refractory tumour which arises from the epithelium of diverse anatomical sites such as oesophagus, head and neck, lung and skin. Accumulating evidence has revealed a number of genomic, clinical and molecular features commonly observed in SCC of distinct origins. Some of these genetic events culminate in fostering the activity of ΔNp63, a potent oncogene which exerts its pro-tumourigenic effects by regulating specific transcriptional programmes to sustain malignant cell proliferation and survival. In this review, we will describe the genetic and epigenetic determinants underlying ΔNp63 oncogenic activities in SCC, and discuss some relevant transcriptional effectors of ΔNp63, emphasizing their impact in modulating the crosstalk between tumour cells and tumour microenvironment (TME).

Keywords: p63; squamous cell carcinoma; transcription factor; tumour microenvironment.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Carcinoma, Squamous Cell / genetics
  • Carcinoma, Squamous Cell / metabolism
  • Carcinoma, Squamous Cell / pathology
  • Epigenesis, Genetic*
  • Gene Deletion*
  • Gene Expression Regulation, Neoplastic*
  • Humans
  • Transcription Factors / deficiency*
  • Transcription Factors / metabolism
  • Tumor Microenvironment*
  • Tumor Suppressor Proteins / deficiency*
  • Tumor Suppressor Proteins / metabolism

Substances

  • TP63 protein, human
  • Transcription Factors
  • Tumor Suppressor Proteins