Cordycepin induces apoptosis in human bladder cancer cells via activation of A3 adenosine receptors

Tumour Biol. 2017 Jul;39(7):1010428317706915. doi: 10.1177/1010428317706915.

Abstract

Bladder cancer is a neoplasm originated from bladder epithelial cells. The therapy for bladder cancer is so far not satisfactory. In this study, we examined the effects of Cordyceps militaris hot water extracts containing cordycepin on human bladder cells. Cordyceps militaris hot water extracts containing cordycepin was used to treat human T24 bladder carcinoma cells, and we found that Cordyceps militaris hot water extracts containing cordycepin decreased T24 cell survival in a dose-dependent manner, which was seemingly mediated by activation of A3 adenosine receptor and the subsequent inactivation of Akt pathways, resulting in increases in cleaved Caspase-3 and apoptosis. Overexpression of A3 adenosine receptor in T24 cells mimicked the effects of Cordyceps militaris hot water extracts, while A3 adenosine receptor depletion abolished the effects of Cordyceps militaris hot water extracts containing cordycepin. Together, these data suggest that Cordyceps militaris hot water extracts containing cordycepin may be a promising treatment for bladder cancer via A3 adenosine receptor activation.

Keywords: Akt; Bladder cancer; apoptosis; cordycepin.

MeSH terms

  • Apoptosis / drug effects*
  • Caspase 3 / genetics
  • Cell Line, Tumor
  • Cell Proliferation / drug effects
  • Cell Survival / drug effects
  • Cordyceps / chemistry
  • Deoxyadenosines / administration & dosage*
  • Deoxyadenosines / chemistry
  • Gene Expression Regulation, Neoplastic / drug effects
  • Humans
  • Oncogene Protein v-akt / genetics
  • Plant Extracts / administration & dosage
  • Plant Extracts / chemistry
  • Receptor, Adenosine A3 / genetics*
  • Signal Transduction / drug effects
  • Urinary Bladder Neoplasms / drug therapy*
  • Urinary Bladder Neoplasms / genetics
  • Urinary Bladder Neoplasms / pathology

Substances

  • Deoxyadenosines
  • Plant Extracts
  • Receptor, Adenosine A3
  • Oncogene Protein v-akt
  • Caspase 3
  • cordycepin