Methylation of MGMT promoter does not predict response to temozolomide in patients with glioblastoma in Donostia Hospital

Sci Rep. 2020 Oct 28;10(1):18445. doi: 10.1038/s41598-020-75477-9.

Abstract

O6-methylguanine-DNA methyltransferase (MGMT) promoter methylation status has been considered a prognostic factor in newly diagnosed glioblastoma (GBM). In this study, we evaluated the prognostic and predictive value of MGMT promoter methylation in patients with glioblastoma in Donostia Hospital. Surprisingly, methylation of MGMT promoter did not predict response to temozolomide in patients with glioblastoma in Donostia Hospital. Specifically, overall survival (OS) and progression-free survival (PFS) did not differ significantly by MGMT methylation status in our cohort. In contrast, both were longer in patients who received treatment, received more TMZ cycles, had a better general status and perform at least a partial resection. No association was detected between methylation of MGMT promoter and molecular markers such as ATRX, IDH, p53 and Ki67. These results indicate that MGMT methylation did not influence in patient survival in our cohort.

Publication types

  • Clinical Trial
  • Observational Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aged
  • DNA Methylation / drug effects*
  • DNA Modification Methylases / metabolism*
  • DNA Repair Enzymes / metabolism*
  • DNA, Neoplasm / metabolism*
  • Disease-Free Survival
  • Female
  • Glioblastoma* / drug therapy
  • Glioblastoma* / enzymology
  • Glioblastoma* / mortality
  • Glioblastoma* / pathology
  • Hospitals
  • Humans
  • Male
  • Middle Aged
  • Predictive Value of Tests
  • Promoter Regions, Genetic*
  • Retrospective Studies
  • Survival Rate
  • Temozolomide / administration & dosage*
  • Tumor Suppressor Proteins / metabolism*

Substances

  • DNA, Neoplasm
  • Tumor Suppressor Proteins
  • DNA Modification Methylases
  • MGMT protein, human
  • DNA Repair Enzymes
  • Temozolomide