1-Methyl-4-phenylpyridinium (MPP+) inhibits mitochondrial oxygen consumption mediated by succinate as well as malate in rat pheochromocytoma PC12 cells

Biochem Mol Biol Int. 1995 Feb;35(2):409-13.

Abstract

When rat pheochromocytoma PC12 cells are cultured with 1 mM 1-methyl-4-phenylpyridinium (MPP+), the number of viable cells decreases to one third in 4 days while the number increases ten-fold without MPP+. Oxygen consumption by mitochondria in the presence of malate is inhibited about 80% by the treatment of the cells with MPP+ for 4 days. Unexpectedly, succinate-dependent oxygen consumption is also inhibited to essentially the same extent as malate-dependent one. These results suggest that the impairment of the respiration mediated by succinate as well as malate is important as a mechanism of MPP(+)-induced cell death.

Publication types

  • Comparative Study

MeSH terms

  • 1-Methyl-4-phenylpyridinium / pharmacology*
  • Adrenal Gland Neoplasms
  • Animals
  • Cell Survival / drug effects
  • Dose-Response Relationship, Drug
  • Kinetics
  • Malates / metabolism*
  • Mitochondria / drug effects
  • Mitochondria / metabolism*
  • Oxygen Consumption / drug effects*
  • PC12 Cells
  • Pheochromocytoma
  • Rats
  • Succinates / metabolism*
  • Time Factors

Substances

  • Malates
  • Succinates
  • 1-Methyl-4-phenylpyridinium