The Th17/Treg Immune Imbalance in Ulcerative Colitis Disease in a Chinese Han Population

Mediators Inflamm. 2016:2016:7089137. doi: 10.1155/2016/7089137. Epub 2016 Feb 9.

Abstract

Objective: To investigate the Th17/Treg immune balance in the ulcerative colitis (UC) patients in a Chinese Han population.

Methods: Ninety UC patients and 30 healthy subjects were enrolled. The serum IL-17 and TGF-β1 levels of these participants were measured with ELISA; the percentage of Th17 and Treg cells in peripheral blood was determined with flow cytometry.

Results: In UC patients, the levels of IL-17 and Th17 were significantly higher compared with healthy subjects; the percentage of Th17 and IL-17 level in moderate and severe subgroup was significantly higher than in mild subgroup; a positive correlation existed between these two indexes and clinical activity index and endoscopic evaluation. TGF-β1 level and Treg cells in UC patients were lower than healthy subjects. TGF-β1 level in moderate and severe subgroup was lower than in mild subgroup. There was a negative linear correlation between Treg cells and clinical activity index, endoscopic evaluation. A positive correlation was detected between Treg cells and TGF-β1 level.

Conclusions: Th17/Treg immune imbalance might play a crucial role in the development of UC. To induce the production of Treg cells and TGF-β1, inhibit the level of Th17 and IL-17, and thus recover the Th17/Treg immune balance might imply new therapeutic targets in UC management.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Adult
  • Aged
  • Colitis, Ulcerative / immunology*
  • Colitis, Ulcerative / metabolism*
  • Enzyme-Linked Immunosorbent Assay
  • Female
  • Flow Cytometry
  • Humans
  • Interleukin-17 / metabolism
  • Male
  • Middle Aged
  • T-Lymphocytes, Regulatory / immunology*
  • T-Lymphocytes, Regulatory / metabolism
  • Th17 Cells / immunology*
  • Th17 Cells / metabolism*
  • Transforming Growth Factor beta1 / metabolism
  • Young Adult

Substances

  • Interleukin-17
  • Transforming Growth Factor beta1