http://rdf.ncbi.nlm.nih.gov/pubchem/patent/ES-2339097-B1

Outgoing Links

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assignee http://rdf.ncbi.nlm.nih.gov/pubchem/patentassignee/MD5_5b4c6f22aa3eb607415018501713d26e
classificationCPCInventive http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/A61K38-18
classificationIPCInventive http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61K38-18
filingDate 2009-11-19^^<http://www.w3.org/2001/XMLSchema#date>
grantDate 2011-02-28^^<http://www.w3.org/2001/XMLSchema#date>
inventor http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_e6df3d06d9dfc8483a6f02c046a6f1d0
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_0ed62e102ba4096c6a5db5b01b7cf021
publicationDate 2011-02-28^^<http://www.w3.org/2001/XMLSchema#date>
publicationNumber ES-2339097-B1
titleOfInvention IMPROVEMENTS IN THE OBJECT OF THE MAIN PATENT N. P200702172, BY "PLEITROPHIN (PTN) AND MIDKINA (MK) FOR THE TREATMENT OF CONSUMPTION ADDICTION AND TOXICITY PRODUCED BY ABUSE DRUGS.
abstract Improvements in the subject of main patent No. P 200702172 by: "Pleitrofin (PTN) and Midkina (MK) for the treatment of drug addiction and drug abuse."n n n Addition to invention patent numbernP200702172, in which the use of cytokines Pleiotrophin (PTN) andnMidkina (MK) in the preparation of medicines for treatmentnof the toxicity produced in humans by drug use ofnabuse, extends to medications for the treatment of addiction orndependence on that consumption, also characterized this newntherapeutic functionality by overexpressionnsignificant of these cytokines that takes place in various areasnof the central nervous system in response to the administration ofndrugs of abuse, which now occurs as a defense mechanismnEndogenous limiter of the addictive effects of these drugs. Thesencytokines can be used alone or in combination of both and / or withnother agents, such as some of the mediating proteins of theirneffects.
priorityDate 2007-08-02^^<http://www.w3.org/2001/XMLSchema#date>
type http://data.epo.org/linked-data/def/patent/Publication

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