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Journal Article|Research Support, N.I.H., Extramural |
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1252 |
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2163-0755
2163-0763 |
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6 |
| pageRange |
1247-1252 |
| publicationName |
The journal of trauma and acute care surgery |
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1247 |
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| bibliographicCitation |
Sadjadi J, Strumwasser AM, Victorino GP. Endothelial cell dysfunction during anoxia-reoxygenation is associated with a decrease in adenosine triphosphate levels, rearrangement in lipid bilayer phosphatidylserine asymmetry, and an increase in endothelial cell permeability. J Trauma Acute Care Surg. 2019 Aug 29;87(6):1247–52. doi: 10.1097/ta.0000000000002489. |
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http://rdf.ncbi.nlm.nih.gov/pubchem/author/MD5_831977897a34e3b21fc87d84b592108b |
| date |
2019-08-29^^<http://www.w3.org/2001/XMLSchema#date> |
| identifier |
https://pubmed.ncbi.nlm.nih.gov/31464867
https://pubmed.ncbi.nlm.nih.gov/PMC6878159
https://doi.org/10.1097/ta.0000000000002489 |
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| language |
English |
| source |
https://pubmed.ncbi.nlm.nih.gov/
https://www.crossref.org/ |
| title |
Endothelial cell dysfunction during anoxia-reoxygenation is associated with a decrease in adenosine triphosphate levels, rearrangement in lipid bilayer phosphatidylserine asymmetry, and an increase in endothelial cell permeability |
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